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August 15, 2025

Anemoside B4 mitigates endoplasmic reticulum stress-induced apoptosis post cerebral ischemia/reperfusion injury in rats.

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Authors

XHXiaohuan HuangRDRan DengCMCongcong Ma

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Overview

Observational analysis shows reduced neuronal apoptosis in male rats with cerebral ischemia, indicating anemoside B4's potential role in neuroprotection through ER stress inhibition.

Key Points

  • AB4 significantly reduces neuronal apoptosis and mitigates damage in cerebral ischemia/reperfusion injury models.
  • Infarct volume decreased significantly following AB4 treatment, demonstrating its protective effects on brain tissue.
  • Assessment using histopathological analysis reveals a decrease in key ER stress markers with AB4 treatment, particularly GRP78.
  • Potential therapeutic applications exist for AB4 in treating conditions related to endoplasmic reticulum stress and neuronal damage.

Cite This Study

Huang et al. (2025) studied this question.

synapsesocial.com/papers/68c235ccb210217d64771dd2https://doi.org/10.22038/ijbms.2025.85441.18475
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