September 10, 2025PeerJOpen Access

Rnf32 is not essential for spermatogenesis and male fertility in mice

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Knockout analysis shows normal fertility outcomes in rnf32 -/- males, highlighting gene's non-essential role.

Male rnf32 knockout mice exhibited normal fertility, demonstrating no significant differences in reproductive outcomes.
Sperm motility, testicular histology, and spermatogenesis were unaffected by the absence of rnf32 in knockout mice.
Utilizing crispr/cas9 technology, rnf32 knockout mice were successfully created, enabling detailed genetic analysis.
These findings suggest a non-essential role for rnf32 in male fertility, prompting further research into its functions.

Kong et al. (2025) studied this question.

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