Design
Review
Key result
The metainflammation-mitochondrial dysfunction axis and organokine-mediated crosstalk drive a self-perpetuating cycle of metabolic dysfunction and cardiovascular disease progression in Metabolic Syndrome.
Authors
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May inform future organokine-targeted therapies in metabolic syndrome; leaves open whether modulating metainflammation improves cardiovascular outcomes.
This review provides an integrated overview of how metainflammation, mitochondrial dysfunction, and organokine crosstalk drive metabolic dysfunction and cardiovascular disease progression in Metabolic Syndrome.
Sodré et al. (2026) conducted a review in Metabolic Syndrome (MetS). Metainflammation and mitochondrial dysfunction was evaluated. The metainflammation-mitochondrial dysfunction axis and organokine-mediated crosstalk drive a self-perpetuating cycle of metabolic dysfunction and cardiovascular disease progression in Metabolic Syndrome.