This review demonstrates metabolic reprogramming influences immune dysfunction and oxidative stress in COPD, suggesting new insights into pathogenesis.
Key Points
Metabolic reprogramming contributes to chronic inflammation and immune dysfunction in COPD.
Findings show key metabolic pathways like lipid, glucose, and amino acid metabolism are involved.
Analysis of immune cells from COPD experimental models reveals environmental stress impacts pathogenesis.
Understanding these interactions may lead to novel therapeutic strategies for COPD management.