November 30, 2025Frontiers in Insect ScienceOpen Access

The GPCR A35 regulates fecundity of Nilaparvata lugens Stål via juvenile hormone signaling

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Fecundity declined by 51.3% due to RNAi-mediated silencing of GPCR A35, impacting juvenile hormone levels.
Silencing GPCR A35 caused a 14.8% reduction in juvenile hormone titer, showing its role in reproductive health.
Analysis involved expression profiling of GPCR A35 in females, and its silencing revealed key effects on hormone signaling.
Findings suggest potential for RNAi-based strategies in managing Nilaparvata lugens infestations, benefiting rice yields.

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RNAi lowered juvenile hormone levels and reduced fecundity by 51% in Nilaparvata lugens, suggesting GPCR A35 as a target for pest control.

Minjuan et al. (2025) studied this question.

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RNAi lowered juvenile hormone levels and reduced fecundity by 51% in Nilaparvata lugens, suggesting GPCR A35 as a target for pest control.