Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
August 21, 2025Acta Odontologica ScandinavicaOpen Access

Advanced glycation end products induce inflammaging in periodontal ligament fibroblasts through RAGE/AKT/mTOR/glycolysis pathway

View Full Paper
Ask AI
Bookmark
Share

Authors

LXLin XiongJSJiangzheng ShuHGHongli Gao

Discussion

Loading...

Member takes

Overview

This research reveals AGEs increase pro-inflammatory cytokines in human periodontal ligament fibroblasts, suggesting RAGE/AKT/mTOR involvement in inflammaging.

Key Points

  • AGEs significantly elevate pro-inflammatory cytokines in response to LPS, linking them to inflammaging.
  • Inhibiting glycolysis mitigates AGE-induced inflammaging markers, indicating the pathway's vital role in inflammation.
  • Blocking the AKT/mTOR pathway suppresses AGE-induced inflammaging and glycolysis, highlighting multiple intervention targets.
  • Antagonizing RAGE effectively prevents AGE-induced inflammation, providing insights for periodontitis treatment.

Cite This Study

Xiong et al. (2025) studied this question.

synapsesocial.com/papers/68af6bff7567bf4f94fe9916https://doi.org/10.2340/aos.v84.44581
View Full Paper
Ask AI
Bookmark
Share